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Potent antioxidant function at the expense of central carbon metabolism The dependency of NRF2-addicted cancer cells on exogenous glutamine is attributable to the elevated consumption and excretion of glutamate as a result of two major transcriptional outputs of NRF2 (Fig

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Kielgast, U., Krarup, T., Holst, J

The rationale is mechanistic rather than proven: tirzepatide activates GLP-1 and GIP receptors while glutathione acts as the body's principal antioxidant, and the assumption is that the two might complement each other

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