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The question: does sustained elevation of intracellular NAD+ via NNMT inhibition produce the same long-term benefits as NAD+ precursor supplementation (like nicotinamide riboside or NMN), or are there trade-offs we haven't identified yet

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This isn't just a "fat burner." Standardized green tea extract operates on multiple pathways: Enhances mitochondrial biogenesis (cellular energy production) Improves glucose uptake and insulin signaling Modulates AMPK pathways for metabolic flexibility For formulators, this represents an evolution

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