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found that restoring GSH levels alleviated hepatic ferroptosis and oxidative stress by upregulating PI3K/Akt/Nrf2 signaling and enhancing the SLC7A11/GPX4 axis

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Mice that received zinc were resistant to cyclophosphamide-induced reductions in numbers of white blood cells and mature T lymphocytes, suppression of IgM antibody production, and reduction of thymus weight (Zhang 1992)