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mTORC2 influences CRC metabolism through AKT hyperactivation and c-Myc upregulation, linking oncogenic signaling to metabolic adaptation [64]
Monomeric C-reactive protein a feature of inflammatory disease associated with cardiovascular pathophysiological complications
3,4 It is known to be antimelanogenic, thereby associated with melanin production
The signature across MS and both demyelination models constituted strong suppression of the homeostatic microglial transcriptional program, including downregulation of P2RY12 , CX3CR1 , ENTPD1 ( CD39 ), BACH2, MEF2A and SALL1