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In addition, markers of liver fibrosis, including hyaluronic acid (HA), type III collagen (PC III), and laminin (LN), provide insights into the extent of extracellular matrix remodeling and fibrosis [12, 13]
Furthermore, FMO3 has been shown to serve as a central regulator of tissue cholesterol balance and to be involved in insulin resistance and dyslipidemic changes in mouse models, suggesting the FMO3 and TMAO pathway as potential mediators of diabetes associated atherosclerosis 52,53
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