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Notably, research using a murine MRSA pneumonia model suggests that inducing MRSA death via ferroptosis characteristics can enhance mouse survival rates, reduce bacterial loads, and mitigate inflammatory damage, offering promising clinical therapeutic implications (Hu et al., 2023a)

The implementation questions and corresponding responses from the clinical experts consulted by CADTH are summarized in Table 7

Pinchas Cohen's laboratory at USC, MOTS-c demonstrated that mitochondrial DNA could produce peptides with systemic effects throughout the body

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Another contributing factor can be a minor histamine release at the injection site