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Although targeting the IL-17A pathway provides a rational approach for corticosteroid-resistant neutrophilic inflammation, early clinical trials using non-selective IL-17A blockade failed to improve patient outcomes
3a), and renders the synthesis of cysteine dependent on the activity of one-carbon metabolism (methionine and folate cycles)
MatsuzawaDObataTShirayamaYNonakaHKanazawaYYoshitomeEet al
This detoxification mechanism has made GSH a critical compound in hepatocyte and toxicology research models
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