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Similar findings in ME/CFS patients suggest that micro-clots and platelet hyperactivation could also play a role in the pathology of this condition, potentially driven by elevated oxidative stress (Nunes et al., 2022)
10.1016/j.drugalcdep.2007.04.004 40 OkanoM.SatoM.KageyamaS
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Homocysteine-lowering vitamins do not lower plasma S-adenosylhomocysteine in older people with elevated homocysteine concentrations
Antioxidant enzymes, such as glutathione peroxidases and peroxiredoxins, generate glutathione disulfide during the reduction of peroxides such as hydrogen peroxide (H2O2) and organic hydroperoxides (ROOH): 2 GSH + ROOH GSSG + ROH + H2O Other enzymes, such as glutaredoxins, generate glutathione disulfide through thiol-disulfide exchange with protein disulfide bonds or other low molecular mass compounds, such as coenzyme A disulfide or dehydroascorbic acid
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