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Despite that mitochondria are exposed to the generation of oxidant species, the existence of an efficient antioxidant defense system, of which mGSH is a critical component, prevents or repairs oxidative damage generated during normal aerobic metabolism (Mari et al., 2013)

It has also been shown to suppress CCl11 and HMGB1 genes, which are markers associated with aging and neurological, cardiovascular, and immune diseases [2]

Signal Transduction and Redox Signaling : Investigates S-glutathionylation and how it can change enzyme activity and transcriptional control

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