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While CBP/p300 is a well-validated direct interaction partner of NRF2 with prominent functions at enhancers, we report that this interaction is not required for NRF2-dependent NSCLC cell growth, indicating that NRF2 can sustain sufficient transcriptional activity in the absence of CBP/p300 coactivation
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Increased thyroid volume and frequency of thyroid disorders signs in schoolchildren from nitrate polluted area
Role that phosphorylation of GSK3 plays in insulin and Wnt signalling defined by knockin analysis
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