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A., Flatt, P
In ApoE / mice that are given a HFD, the persistent activity of the SNS exacerbates oxidative stress in ECs by increasing blood levels of norepinephrine and the activity of mitochondrial monoamine oxidase A (MAO-A), culminating in mitochondrial dysfunction and inflammatory responses
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High homocysteine is also linked to increased blood clots.[ref][ref][ref][ref] Related article: Homocysteine- Genetics and Solutions 3
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