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While glutathione itself crosses this barrier poorly, the brain can synthesize glutathione from precursor amino acids, and systemic glutathione elevation may indirectly support brain glutathione status through various mechanisms including reduced oxidative stress on blood-brain barrier integrity and improved availability of glutathione precursors

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Notably, simultaneously activating ARC AgRP/NPY neurons and inhibiting POMC neurons additively increases food intake in the acute setting [143]

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