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It's cell study data, not a clinical vascular outcome, but the mechanism it points to is physiologically coherent
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Diabetes Care 35 (3), 556564
In first protocol, the male mice were deprived of food from 18:00 (16 h fasting) with free access to water 1 day before the experiment
18 In kidney injury, extracellular vesicles and secreted lipid mediators are sufficient to synchronize ferroptotic death across spatially contiguous tubular epithelial cells, thereby contributing to a self-amplifying wave of tubular death during the transition from acute kidney injury to chronic kidney disease
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