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Moreover, Adeshakin et al

Their paper, Glutathione peroxidase activity is neuroprotective in models of Huntingtons disease , was published in Nature Genetics

I mean peptides, you know, the basic way to think about them is they're cellular messengers

However, the proteins identified as differentially expressed between non-splenectomized -thalassemia/HbE patients and healthy volunteers included a number of cytoskeletal proteins (F-actin -capping protein subunit beta, myosin light polypeptide 6, myosin regulatory light chain 12A, myosin-9, Rho GDP-dissociation inhibitor 2, transgelin-2, PDZ and LIM domain protein 1, and tropomyosin alpha-4 chain) as well as heat shock protein 70 kDa protein, integrin alpha IIb, beta-2-microglobulin, hemoglobin subunit beta, platelet factor 4 (or chemokine (C-X-C motif) and leukocyte elastase inhibitor

Glutathione-S-transferase selective release of metformin from its sulfonamide prodrug