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For the newer GLP-1 medications, it would be useful to look at whether there are genetic variants, like the variants in PAM, that explain poor responders to their medications. The biological basis of GLP-1 resistance is still unknown and is likely complex

Beyond semaglutide response prediction, the genetic test reveals underlying metabolic vulnerabilities related to hunger signaling, fat storage (FTO variants), and melanocortin pathway function

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But the drug will need to be tested in much bigger trials to assess its effects in larger groups of people

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