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It links the pattern to dose escalation phases specifically, describing cramps as most likely when the amount of medication changes rather than at a steady state
No mechanism of action explains how b12 would reduce nausea caused by delayed gastric emptying
But the mechanism here is plausible enough to warrant attention in human studies too
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Monoacylglycerol binds to GPR119 in L cells to promote the release of GLP-1, GIP and insulin ( 2+ were observed following GPBA-R activation in single GLUTag cell, and bile acid and GPBA-RA increased GLP-1 secretion in intestinal cultures (Parker et al., 2012b)
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