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This medication enhances glucose-dependent insulin secretion from pancreatic beta cells, meaning it stimulates insulin release only when blood glucose levels are elevated, thereby reducing the risk of hypoglycaemia compared to some other diabetes medications

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Fibroblast studies have reported collagen synthesis increases of up to 70% under GHK-Cu stimulation

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Animal models suggest prevention of doxorubicin-induced acute hepatotoxicity occurs via suppression of intrinsic caspase-3-dependent apoptotic signaling (24)