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The pathophysiology of AKI in sepsis is complex and multifactorial, encompassing intrarenal hemodynamic changes, endothelial dysfunction, infiltration of inflammatory cells within the renal parenchyma, and obstruction of renal tubules by necrotic cells and debris (112,113)

Common nutritional gaps that correlate with stalled recovery Low protein intake relative to body size and training stress Low vitamin D status (common in indoor lifestyles) Low magnesium intake, especially with stress, sweating, or poor diet Inadequate vitamin C in those with limited fruit and vegetable intake Mineral imbalance when restrictive dieting is prolonged When people run peptides while under-eating protein, sleeping poorly, and avoiding micronutrient-dense foods, the body has less material for repair

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