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Additionally, GLP-1 therapies suppress glucagon, a hormone that normally elevates blood sugar, further enhancing glucose regulation
They've been built on decades of metabolic research, and a growing body of clinical data supports their effectiveness
Mechanistically, C3G activated the PPAR/--catenin-TCF-4 pathway in STC-1 cells, enhancing PG precursor transcription and GLP-1 synthesis.Inhibiting PPAR/ with GSK0660 blocked this C3G-induced upregulation
Aguera Z, Lozano-Madrid M, Mallorqui-Bague N, Jimenez-Murcia S, Menchon JM, Fernandez-Aranda F
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