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Increasing evidences show that oxidative stress contributes significantly to the development of gastric cancer, either through initiation, promotion, and progression of cancer cells or causing cell death

These include a Section 402 Part D demonstration and a Center for Medicare and Medicaid Innovation demonstration

Pathways provide targeted therapy and a mechanistic explanation, but they are risky if pathways go off balance

A significant limitation of the current clinical evidence is its reliance on post-hoc analyses of cardiovascular outcome trials and large-scale retrospective studies