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Together, they both increase production and enhance release, creating a powerful anabolic and anti-catabolic environment even during caloric restriction
Class Long-acting IGF-1 analog Note Reduced binding-protein affinity IGF-1 Receptor Studied for extended signaling on the IGF-1 pathway
FIGURE 1 3 Excitotoxicity in TBI The disruption of the BBB resulting from TBI leads to the release of excessive neurotransmitters and a breakdown in the normal glutamate reuptake by transporters (Chamoun, Suki, Gopinath, Goodman and Robertson, 2010)
Grabarevic Z, Tisljar M, Artukovic B, Bratulic M, Dzaja P, Seiwerth S, et al
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