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(b-d) Representative subsets of mice from Fig
In healthy tissue, BPC-157 appears to maintain nitric oxide homeostasis by modestly increasing eNOS activity while suppressing iNOS
This table is math, not a dosing recommendation
BPC 157 activated the Src-Cav-1-eNOS signal pathway and reduced eNOS/Cav-1 binding in vascular endothelial cells It is known that phosphorylation of Src can promote Cav-1 activation and subsequent endocytosis of tyrosine kinase receptors and eNOS activation 20,24
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