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Chloroquine (CQ) indirectly inhibits the ubiquitination and degradation of FTH1 mediated by NCOA4 by blocking the fusion of autophagosomes and lysosomes, thereby significantly alleviating ferroptosis-related neuronal damage[222]

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An open-label, randomized trial indicates that everolimus with tacrolimus or cyclosporine is comparable to standard immunosuppression in de novo kidney transplant patients

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