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In the OLZ-injected group, the physiological alterations were represented by the disappearance of two normal types (-EST3 and -EST4)

Morgan, B

Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)

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Figure 5: Acetaldehyde, a byproduct of alcohol, disrupts the balance between SAM and SAH