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Autophagic flux tracing using tandem mRFP-GFP-LC3 probe showed that increase of autophagic flux caused by GSH depletion is augmented by rapamycin and attenuated by autophagic inhibitors 3-MA and Baf-A1 (Fig

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Research indicates that the rs1967017 variant in PDZK1 creates a binding site for the transcription factor hepatocyte nuclear factor 4 (HNF4) within an enhancer region upstream of the PDZK1 transcription start site

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Derived from a protein found naturally in human gastric juice, it is exceptionally stable