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Swanson CA, Patterson BH, Levander OA, Veillon C, Taylor PR, Helzlsouer K et al
In diet-induced obesity models, this inhibition has been observed to reduce white fat accumulation and promote lipolysis, effectively reprogramming metabolic pathways toward enhanced energy expenditure
Furthermore, one study reveals that IGF-1-specific muscle expression counteracts muscle wasting caused by angiotensin II [3]
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