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Based on the demonstration that TRMs require IL15 for their differentiation, the interruption of the IL15 pathway using an anti-CD122 antibody caused reversal of depigmentation in mice with stable vitiligo

7a, b, Cys 2 starvation as well as BSO and erastin treatment increased the percentage of cells in the G0/G1 phase with a concomitant decrease in the G2 phases, suggesting that GSH depletion induces growth arrest in RPE cells

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No new safety signals emerged late in the treatment period that were not present early on