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In post-ischemic rat hearts, treatment with dapagliflozin results in higher level of IL-10, a cytokine that facilitates the conversion of macrophages from a pro-inflammatory M1 to anti-inflammatory M2 phenotype, thereby inhibiting myofibroblast differentiation and extracellular matrix formation (Lee et al., 2017)
doi: 10.1093/ecco-jcc/jjaf010 84 ChanSSMChenYCaseyKOlenOLudvigssonJFCarbonnelFet al
Microplastics in neonates: an overlooked cost of parenteral nutrition
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Furthermore, as seen above, an enhancement of endogenous GHRH release would also take place
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