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Without adequate GSH, the body's antioxidant network functions at reduced capacity
Both enzymatic and non-enzymatic antioxidant systems scavenge and deactivate excessive free radicals, helping to prevent cell damage
Although this is consistent and mechanistically validated across different preclinical models, as reviewed in the Preclinical and clinical applications of NAD + -modulating interventions for mitochondrial homeostasis and health benefits section, the correspondence between NAD + changes and mitochondrial or metabolic alterations is more complex in human studies
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Autophagy exhibits selectivity in degrading certain cargoes such as mitochondria (mitophagy) endoplasmic reticulum (reticulophagy), intracellular microbes (xenophagy), lipid droplets (lipophagy), peroxisomes (pexophagy) and glycogen (glyophagy) or non-selective to degrade random parts of the cytoplasm (144)
