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Thus, we measured TrB1-3 distribution in B cell-specific Gclc -deficient mice and found that TrB1 were increased in Gclc -deficient mice compared to controls (Supplementary Fig
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Examination of actin-binding and cell-migration activity of Thymosin Beta-4 (TB-500) in wound-healing and cardiac-tissue research models, including comparison against single-peptide TB-500 reference material
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In: Proceedings of the 2009 symposium on languages in biology and medicine
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