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Shown to decrease chronic inflammation

Although SNS-derived catecholamines probably remain a primary mediator of the day-to-day regulation of peripheral WAT, once in a state of chronic hypoinsulinaemic hypoglycaemia, we expect that the repertoire of lipolytic agonists expands substantially

1), which is further supported by unaltered mRNA levels of inflammatory genes TNF, IL-6 and IL-1 (supplementary Fig

However, too much of it can overstimulate the process of detoxification, thus leading to detoxification overload