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It has been demonstrated that addressing the deficit in synaptic plasticity caused by neuronal atrophy and cell death can be beneficial in the treatment of depression ( Currently, the primary focus of clinical depression treatment lies in targeting monoamine neurotransmitters
These effects build gradually and may take several weeks to become noticeable
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In a number of studies of patients with MDD, evidence has been found of increased neuroinflammatory markers associated with lower levels of antioxidants (Morris et al
Redness and swelling last 2448 hours a normal healing response
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