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We next sought to evaluate the relationship between the level of PS exposure and axon injury responses by generating heatmaps using average index values of AnxV levels, axonal fusion and debris from wildtype and gpx mutants under different conditions

The core of this method lies in the separate measurement of the wet weight of the brain tissue and its weight after dehydration

[1] discovered a novel form of non-apoptotic cell death characterized by iron overload and accumulation of lethal lipid peroxidation [2]