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[10] have shown that muscle-specific loss of carnitine acetyltransferase (CrAT) in mice led to an accumulation of acetyl-coenzyme A (CoA) within the mitochondria and allosteric inhibition of pyruvate dehydrogenase (PDH), which subsequently impaired glucose utilization

A mechanism associated with increased mitochondrial activity and energy expenditure

Sequential multi-omics analysis identifies clinical phenotypes and predictive biomarkers for long COVID

Mahnaz Momeni, M.D

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