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Association of genetically predicted lipid levels with the extent of coronary atherosclerosis in Icelandic adults
GSH can be exhausted via inhibiting SLC7A11 by traditional drugs (e.g., sorafenib and sulfasalazine (SAS)) or novel compounds (e.g., erastin), thus inducing ferroptosis (Dixon et al., 2012,2014)
Researchers at the University of Minnesota have developed a novel glutathione analog, -GSH, that resists -GT degradation while retaining critical GSH functions
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