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Furthermore, two previously used antischistosomal compounds inhibited TGR activity, suggesting that TGR is a key target during therapy with those compounds
Linagliptin is preferred as it requires no dose adjustment in renal failure
This vulnerability is often amplified in obesity, where baseline oxidative tone is elevated and polyunsaturated fatty acid (PUFA)-enriched membrane phospholipids provide oxidation-susceptible substrates [66,67,68]
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This warning stems from rodent studies that resulted in the animals developing thyroid C-cell tumors
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