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While androgen excess is primarily associated with reproductive pathology, physiological levels of androgens are essential for normal endometrial proliferation and differentiation through receptor-mediated mechanisms [31, 32]
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Similarly, CPUY192018, a potent small-molecule inhibitor of the Keap1-Nrf2 PPI, demonstrated cytoprotective effects in NCM460 colonic cells and a DSS-induced UC model by activating Nrf2 signaling (262)
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