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Due to its polarity, the acetaminophenglucoside elutes earlier than all other acetaminophen-related compounds (RT 1.7 min

recently showed that direct GPx4 overexpression in the cellular models (A549, H460 and H1299 and H23) of non-small cell lung carcinoma (NSCLC) prevented the radiation-induced lipid peroxidation and, in turn, contributed to radioresistance by blocking the ferroptosis mode of cell death [56]

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of Na 2 S 2 followed by trapping with IAM-TPP (Fig