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In conclusion, while substantial advances have elucidated metabolic reprogrammings role in CRC, therapeutic development demands precision strategies addressing pathway complexity and TME crosstalk

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Studies have shown that, in both non-obese diabetic (NOD) mouse models and patients with T1DM, dysregulated arginine metabolism is closely associated with increased macrophage NO production and is linked to pancreatic -cell injury and disease progression (99)

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