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Through substrate competition between iNOS and Arg1, arginine couples inflammatory NO production with polyamine/reparative metabolic branches, thereby regulating the balance between pro-inflammatory cytotoxicity and tissue repair within the same substrate framework ( Taking TCA cycle reprogramming as an example, pro-inflammatory activation is often accompanied by TCA cycle disruption and the accumulation of signaling intermediates, among which succinate has been shown to promote IL-1 production and amplify inflammatory responses through HIF-1 (132)
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"A Chemical Study of the Secretion of the Anal Glands of Mephitis mephitiga (Common Skunk), with Remarks on the Physiological Properties of This Secretion"
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