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Mitochondrial translocase of the outer membrane alterations may underlie dysfunctional oxidative phosphorylation in alzheimer's disease

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Abstract Alzheimers disease (AD) is a progressive neurodegenerative disorder characterized by cognitive decline, memory impairment, and hallmark neuropathological features, including extracellular amyloid- (A) plaques and intracellular neurofibrillary tangles formed by hyperphosphorylated tau

Semaglutide* is a medication that results in changes to the digestive processes, often leading to significant weight loss

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