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Glutamate/GABA imbalance can cause nerve excitotoxicity, leading to nerve cell damage and neurological dysfunction
On top of all of this, GLP-1 might also help us control how much we eat
Under conditions of chronic stress or systemic inflammation, enzymes such as indoleamine 2,3-dioxygenase (IDO) and tryptophan 2,3-dioxygenase (TDO) are upregulated, diverting TRP metabolism away from 5-HT synthesis toward KYN production, with profound consequences for both peripheral physiology and central nervous system (CNS) function
This non-coenzyme action of thiamine in neuronal signaling is further supported by the identified molecular targets of thiamine and its non-coenzyme derivative ThTP
Glucagon-like peptide 1 (GLP-1) secretion and plasma dipeptidyl peptidase IV (DPP-IV) activity in morbidly obese patients undergoing biliopancreatic diversion
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