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its whole logic is to sustain the fullness signal over time, not to deliver a few-week push

This rapid turnover makes it unlikely that peripheral GLP-1 significantly contributes to endogenous activation of central GLP-1Rs

Estrogens signal through myriad pathways, including genomic mechanisms mediated by two nuclear receptors (ER and ER) that recruit various co-regulatory proteins and induce transcription at estrogen response elements (EREs), in addition to other promoters by tethering to transcription factors (e.g., AP-1 and NF-B)

Purpose-built for physicians GLP-1s tied to lower risk for 10 types of cancer compared with insulin in type 2 diabetes Key takeaways: People with type 2 diabetes using a GLP-1 medication have a lower risk for most obesity-associated cancers than insulin users

Diabetes 4 , 190201 (2013)