These findings indicate that even though ethanol may not be required for TNBS-induced damage, this vehicle enhanced the biological action of TNBS in agreement with previous results (Morris et al, 1989), presumably by acting as a barrier breaker and facilitating its interaction with cellular GSH stores (Table 1)
Wang AM, Ma C, Xie ZH, Shen F
Reducing SLC25A40 expression using siRNA destabilized ISC-rich ETC proteins and elevated mitochondrial and cellular reactive oxygen species (ROS)
However, to our knowledge, no studies have comprehensively explored these assumptions
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