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Redox-responsive type The high concentration of reducing agents like glutathione (GSH) within the cytosol, contrasted with the oxidizing environment of lysosomes, offers a biochemical gradient for designing escape mechanisms
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Glutathione depletion renders hepatocytes vulnerable to TNF-alpha-induced apoptosis, contributing to alcoholic hepatitis [6][35]
After seven days of osmotic stress, SBT1.7 was downregulated in all genotypes in experiment 4 (fold changes Eurobravo 0.22, Eurostarch 0.27, Maxi 0.32, Tomba 0.33), as well as in Eurostarch (0.03) and Tomba (0.09) in experiment 5 (Table 3 b)
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