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There is accumulating evidence that the wild type TP53 not only prevents tumorigenesis by regulating the transcription of genes involved in cellular proliferation, DNA repair and cellular death, but is also involved in the regulation of cholesterol metabolism, suggesting that altered cholesterol metabolism may contribute to HGSC development [111]

10.1165/rcmb.2023-0038OC 37 YanqingL.ZhangC.ChengH.LvL.ZhuX.MaM.et al (2024)

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Clinical research on CagriSema has reported metabolic effect sizes that exceed those of either compound alone in similar populations

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