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The review also covered three therapeutic research strategies: direct GSH depletion (leading to ferroptosis or enhanced chemotherapy sensitivity), indirect approaches targeting the tumour microenvironment, and prodrug strategies utilising elevated tumour GSH for drug activation

The mechanism proposed: glutathione's role in reducing systemic inflammation, which otherwise triggers muscle protein breakdown during prolonged caloric deficits

Rather than commanding a single receptor pathway, GHK-Cu appears to act as a copper carrier and a broad signaling molecule

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Impact: Positive Key areas of impact: Level of evidence: Preliminary Level of risk: Negligible How it works GHK-Cu chelates copper ions, enabling the activation of fibroblasts for more intensive production of collagen and glycosaminoglycans