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The active ingredient (GHK peptide) is from the same family, but this study used a specific compound (ALAVAX) that combines GHK with 5-ALA adding a cellular energy component

This can denature (destroy) the peptide at the air-liquid interface

Treatment with FOXO4-DRI at 25 M in serum-limited senescent fibroblast and keloid organ culture models promoted apoptosis, decreased G0/G1 phase cells, and induced p53-pS15 nuclear exclusion confirmed by Annexin V-FITC/PI apoptosis assays and TUNEL staining of keloid organ cultures at days 3, 7, and 10

This aligns with the biology: repeated stimulation of the wound healing cascade and growth factor release compounds over time

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