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Our 42-year-old female patient who was struggling with long-standing hyperpigmented patches of melasma on her cheeks

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and (ii) off-target resistance caused by the activation of bypass signal transduction resulting from the amplification of the ERBB family of receptor tyrosine kinase genes, BRAF amplification, KRAS amplification, and KRAS mutations [148]

If left untreated, the walls will eventually crush in on the cord, making the section practically useless while causing enormous amounts of pain

The practical implications of this distinction are significant